Abstract

Summary

Green light exposure at just 10 lux produced significant analgesic effects in mice, mediated primarily by cone photoreceptors activating enkephalinergic neurons in the ventrolateral geniculate nucleus (vLGN) and projecting to the dorsal raphe nucleus—not through ipRGCs. These findings suggest that tuning lighting spectra toward green wavelengths at low intensities could be a viable, non-pharmacological pain management strategy in healthcare environments.
Abstract

Key Findings

  • Full-field green light at 10 lux produced significant analgesic effects in both healthy mice and an arthrosis pain model.
  • Cone photoreceptor ablation completely abolished the analgesic effect; rod ablation only partially reduced it; ipRGC ablation had no effect.
  • Inhibition of the retino-vLGN pathway completely abolished green light analgesia.
  • Knockdown of proenkephalin (Penk) in the vLGN or ablation of vLGN-Penk neurons prevented green light analgesia.
  • Activation of vLGN-Penk projections to the dorsal raphe nucleus (DRN) was sufficient to suppress nociceptive behaviors, while inhibition of this projection abolished analgesia.
Categories

Categories

The Science of Light: Identifies specific photoreceptor types (cones, rods, ipRGCs) and neural circuits mediating green light analgesia, clarifying the mechanistic basis of spectral light effects.
Patient Recovery: Green light exposure at low intensity produced measurable analgesic effects relevant to non-pharmacological pain management in clinical settings.
Authors

Author(s)

YL Tang, AL Liu, SS Lv, ZR Zhou, H Cao
Publication Date

Publication Year

2022
Citations

Number of Citations

6
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